Anaerofustis stercorihominis
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Its ID gene: the stretch scientists read to tell microbes apart. Each bar is one letter of its DNA. Every species has its own order of letters, so the pattern works like a fingerprint. Close relatives share most of it.
16S rRNA gene, V4 region: the 253 letters between the standard primers (515F/806R), which are the same in every species. One reference sequence; strains can differ by a letter or two. NCBI NR_027562.1
Small for a gut microbe: longer than 21% of the 2,354 we have genomes for.
33% G+C, higher than 8% of gut microbes.
To scale: if its ID gene were 1 cm long, its genome would stretch 15 meters. Yours would stretch 21 kilometers. One reference genome; strains vary. NCBI GCF_000154825.1
What it can do
- Can make butyrate A short-chain fatty acid that feeds the cells lining the colon.
- Mixed evidence Can use lactose The sugar in milk and dairy. Genome analysis says yes; a lab test on 1 strain says no.
- Can change bile acids Modifies the bile acids the liver makes to digest fat.
Not seen in its genomes or lab tests: make propionate, make lactic acid, ferment fiber, eat inulin, eat resistant starch, eat pectin, feed on gut mucus, make B vitamins, make hydrogen sulfide, make methane.
All 13 capabilities and their evidence
| Capability | Verdict | Genome analysis | Lab tests | Published research |
|---|---|---|---|---|
| Can make butyrate | Can | 1 of 1 genome | — | No papers |
| Can make propionate | Not seen | 0 of 1 genome | — | No papers |
| Can make lactic acid | Not seen | 0 of 1 genome | — | No papers |
| Can ferment fiber | Not seen | 0 of 1 genome | 0 of 1 strain · strip test | No papers |
| Can eat inulin | Not seen | 0 of 1 genome | — | No papers |
| Can eat resistant starch | Not seen | 0 of 1 genome | — | No papers |
| Can eat pectin | Not seen | 0 of 1 genome | — | No papers |
| Can use lactose | Mixed evidence | 1 of 1 genome | 0 of 1 strain | No papers |
| Can feed on gut mucus | Not seen | 0 of 1 genome | — | No papers |
| Can change bile acids | Can | 1 of 1 genome | — | No papers |
| Can make B vitamins | Not seen | 0 of 1 genome | — | No papers |
| Can make hydrogen sulfide | Not seen | 0 of 1 genome | — | No papers |
| Can make methane | Not seen | 0 of 1 genome | — | No papers |
AGORA2 (Heinken et al. 2023) via VMH, CC BY 4.0 · BacDive (DSMZ), CC BY 4.0 · NJC19 (Lim et al., Scientific Data 2020), CC0
In healthy guts
Found in 10.0% of healthy people.
- largest Segatella copri 6.2%
- 2nd largest Phocaeicola vulgatus 5.0%
- 3rd largest Faecalibacterium prausnitzii 4.7%
- 4th largest Bacteroides uniformis 3.2%
- 5th largest Agathobacter rectalis 2.5%
- 6th largest Alistipes putredinis 2.3%
- 7th largest Bifidobacterium longum 2.1%
- 8th largest Ruminococcus bromii 1.8%
- Anaerofustis stercorihominis under 0.01%
- 2,169 other microbes, each a sliver 56.7%
- 15.6%
On average, a sliver, under 0.01% of the microbes in a healthy gut.
Every group, drawn the same way. Only some samples record age or sex, so each group says how many people it comes from. Tap one to see it above.
In conditions
Compared with healthy people in the same studies, it’s less common in 1 condition and more common in 3 conditions. Only studies that had both are counted.
A difference is an association: it doesn’t mean this microbe causes, prevents or treats any of these.
Strongest first, then the biggest gap · Each figure is 10 peopleIn studies of how people live
“Higher” is the study’s measure: on average, a bigger share of gut bacteria across everyone in the group, carriers or not. It isn’t how many people carry it. An association the authors report, measured once per person. It doesn’t mean a diet changes this microbe, or that the microbe affects health.